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NEUROLOGY 2006;67:1482-1485
© 2006 American Academy of Neurology
Brief Communications

Hepatitis C virus infection and neurocognitive function

M. Soogoor, MD, H. S. Lynn, PhD, S. M. Donfield, PhD, E. Gomperts, MD, T. S. Bell, PhD, E. S. Daar, MD for the Hemophilia Growth and Development Study*

From the Los Angeles Biomedical Research Institute at Harbor-UCLA Medical Center, Divisions of HIV Medicine and Infectious Diseases, Department of Medicine, David Geffen School of Medicine at UCLA, Los Angeles, CA (M.S., E.S.D.); School of Public Health, Fudan University, Shanghai, China (H.S.L.); Rho, Inc., Chapel Hill, NC (S.M.D.); Children's Hospital Los Angeles, Los Angeles, CA (E.G.); and Alliant International University, Los Angeles, CA (T.S.B.).

Address correspondence and reprint requests to Dr. Eric S. Daar, Division of HIV Medicine, Harbor-UCLA Medical Center; 1124 West Carson Street, N-24, Torrance, CA 90502; e-mail: EDaar{at}LABioMed.org

Hepatitis C virus (HCV) infection may be associated with neurocognitive deficits. The Hemophilia Growth and Development Study enrolled HIV-infected and HIV-uninfected patients and a group of nonhemophiliac siblings. After controlling for multiple factors, HCV monoinfection was not associated with deficits in adaptive behavior, intelligence, or attention/concentration.

*Participating centers and individuals are listed in the appendix.

The work was funded with support from 1 R01 HD41224, AI43638, and AI27660 from the NIH and CCTG-CC99 SD003 from the Universitywide AIDS Research Program. HIV-1 RNA and hepatitis C virus RNA measurements were performed by Bayer Healthcare, Diagnostics.

Disclosure: The authors report no conflicts of interest.

Received October 4, 2005. Accepted in final form June 23, 2006.






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