Neurology
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
QUICK SEARCH:   [advanced]


     

This Article
Right arrow Full Text (PDF)
Right arrow Correspondence:
Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when Correspondence are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Zwick, D.
Right arrow Articles by Roessmann, U.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Zwick, D.
Right arrow Articles by Roessmann, U.
NEUROLOGY 1985;35:891
© 1985 American Academy of Neurology

Central spinal myelinolysis

David Zwick, MD, Reinhard L. Friede, MD and Uros Roessmann, MD

Institute of Pathology (Dr. Zwick) and the Division of Neuropathology (Dr. Roessmann), Case Western Reserve University, Cleveland, OH; and the Department of Neuropathology (Dr. Friede), University of Göttingen, Göttingen, Federal Republic of Germany.

A midline lesion of the funiculus gracilis was found in four patients with histories of alcoholism, nutritional disturbance, and repeated episodes of electrolyte imbalance. Histologic and anatomic abnormalities were similar to those of central pontine myelinolysis. Sodium imbalance may play a role in the pathogenesis of both conditions.

Address correspondence and reprint requests to Dr. Roessmann, Division of Neuropathology, Institute of Pathology, Case Western Reserve University, 2085 Adelbert Road, Cleveland, OH 44106.

Accepted for publication October 3, 1984.






HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 1985 by AAN Enterprises, Inc.