Neurology
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
QUICK SEARCH:   [advanced]


     

This Article
Right arrow Full Text (PDF)
Right arrow Correspondence:
Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when Correspondence are posted
Right arrow Alert me if a correction is posted
Right arrow Citation Map
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via HighWire
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by Sunohara, N.
Right arrow Articles by Satoyoshi, E.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Sunohara, N.
Right arrow Articles by Satoyoshi, E.
NEUROLOGY 1984;34:321
© 1984 American Academy of Neurology

Action-induced rhythmic dystonia

An autopsy case

Nobuhiko Sunohara, MD, Masakuni Mukoyama, MD, Yukio Mano, MD and Eijiro Satoyoshi, MD

From the National Center for Nervous, Mental and Muscular Disorders, Tokyo, Japan.

We studied a patient with action-induced rhythmic dystonia that followed a stroke. Postmortem studies showed an infarct in the right posterolateral ventral part of the thalamus. Electrophysiologic analysis indicated that the eliciting factor of the involuntary movement was an impulse, promoting voluntary contraction of muscle. CSF 5-HIAA content was low, and HVA was high. Administration of 5-HTP and clonazepam abolished the involuntary movements.

Address correspondence and reprint requests to Dr. Sunohara, National Center for Nervous, Mental and Muscular Disorders, 2620, Ogawa-Higashi-Machi, Kodaira, Tokyo, Japan, 187.

Accepted for publication June 16, 1983.




This article has been cited by other articles:


Home page
 J. Neurol. Neurosurg. PsychiatryHome page
C Chuang, S Fahn, and S J Frucht
The natural history and treatment of acquired hemidystonia: report of 33 cases and review of the literature
J. Neurol. Neurosurg. Psychiatry, January 1, 2002; 72(1): 59 - 67.
[Abstract] [Full Text] [PDF]

Home page
 NeurologyHome page
S. Lehericy, S. Grand, P. Pollak, F. Poupon, J.-F. Le Bas, P. Limousin, P. Jedynak, C. Marsault, Y. Agid, and M. Vidailhet
Clinical characteristics and topography of lesions in movement disorders due to thalamic lesions
Neurology, September 25, 2001; 57(6): 1055 - 1066.
[Abstract] [Full Text] [PDF]

Home page
 J. Neurol. Neurosurg. PsychiatryHome page
S Shirane, M Sasaki, D Kogure, H Matsuda, and T Hashimoto
Increased ictal perfusion of the thalamus in paroxysmal kinesigenic dyskinesia
J. Neurol. Neurosurg. Psychiatry, September 1, 2001; 71(3): 408 - 410.
[Abstract] [Full Text] [PDF]

Home page
 BrainHome page
J. S. Kim
Delayed onset mixed involuntary movements after thalamic stroke: Clinical, radiological and pathophysiological findings
Brain, February 1, 2001; 124(2): 299 - 309.
[Abstract] [Full Text] [PDF]

Home page
 Arch NeurolHome page
M. Vidailhet, C. Dupel, S. Lehericy, P. Remy, D. Dormont, M. Serdaru, P. Jedynak, H. Veber, Y. Samson, C. Marsault, et al.
Dopaminergic Dysfunction in Midbrain Dystonia: Anatomoclinical Study Using 3-Dimensional Magnetic Resonance Imaging and Fluorodopa F 18 Positron Emission Tomography
Arch Neurol, August 1, 1999; 56(8): 982 - 989.
[Abstract] [Full Text] [PDF]


HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 1984 by AAN Enterprises, Inc.