Neurology
HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
 QUICK SEARCH:   [advanced]


     


This Article
Right arrow Full Text (PDF)
Right arrow Correspondence:
Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when Correspondence are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Download to citation manager
Right arrow reprints & permissions
Citing Articles
Right arrow Citing Articles via Google Scholar
Google Scholar
Right arrow Articles by WHITE, D. N.
Right arrow Articles by STEVENSON, R. J.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by WHITE, D. N.
Right arrow Articles by STEVENSON, R. J.
NEUROLOGY 1976;26:683
© 1976 American Academy of Neurology

Transient variations in the systolic pulsations in amplitude of intracranial echoes

Their artifactual origin

D. N. WHITE, M.D. and R. J. STEVENSON

From the Department of Neurology, Queen's University, Kingston, Ontario.

Variations in the amplitude of intracranial echoes that synchronize with systole are caused by movement of the reflecting interfaces. These movements result from propagation of the arterial pulse through the brain and vary with the arterial pulse pressure and the distance through the vascular tree for which the pulse propagates before being completely attenuated; this distance varies with the degree of arteriolar dilatation. The interpretation of such systolic fluctuations in echo amplitudes is subject to a number of limitations, and their recording is also subject to artifactual variations, usually resulting from the relative motion of the signal with respect to a fixed gate. Transient variations in the systolic fluctuations of echo amplitudes, as a result of various stimuli, can be recorded by a fixed gate but not simultaneously by a tracking gate. They would therefore appear to be artifactual and not indicative of regional changes in cerebral blood flow, which in any case would cause motion and amplitude changes in interfaces that were not confined to the region involved.

Received for publication September 19, 1975.

Dr. White's address is Department of Neurology, Queen's University, Kingston, Ontario, Canada K71 3N6.







HOME HELP FEEDBACK SUBSCRIPTIONS ARCHIVE SEARCH TABLE OF CONTENTS
Copyright © 1976 by AAN Enterprises, Inc.